Comparative molecular analysis of therapy-related and de novo acute promyelocytic leukemia.

نویسندگان

  • T Ottone
  • L Cicconi
  • S K Hasan
  • S Lavorgna
  • M Divona
  • M T Voso
  • E Montefusco
  • L Melillo
  • E Barragán
  • U Platzbecker
  • L Giannì
  • M Hubmann
  • M Pagoni
  • S Amadori
  • F Lo-Coco
چکیده

Therapy-related acute promyelocytic leukemia (t-APL) has been reported as a late complication of exposure to radiotherapy and/or chemotherapeutic agents targeting DNA topoisomerase II. We have analyzed in t-APL novel gene mutations recently associated with myeloid disorders. Unlike previous reports in acute myeloid leukemia (AML), our results showed neither IDHs nor TET2 mutations in t-APL. However we found an R882H mutation in the DNMT3A gene in a patient with t-APL suggesting a possible role of this alteration in the pathogenesis of t-APL.

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عنوان ژورنال:
  • Leukemia research

دوره 36 4  شماره 

صفحات  -

تاریخ انتشار 2012